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From the outset SAGE (no immunologists/ virologists present) and the modelling of the theoretical physicist (not an immunologist) Neil Ferguson have worked from the assumption that SARS-CoV-2 is a totally new (novel) virus. They therefore assume that no pre-existing community immunity exists. Hence the worldwide coordinated response of suppression until vaccination through physical interventions (lockdown, masks & distancing).
SARS-CoV-2 is a Coronavirus, which are a related group of RNA viruses that cause disease in mammals and birds. Pre-exisiting community immunity already exists for Coronaviruses. That is why the vast majority of deaths attributed to SARS-CoV-2 are from people with weak immune systems (75+years old with 2 or more comorbities). This piece of the pathology of SARS-CoV-2 is now known.
With the new Wuhan asymptomatic antibody study another piece of pathology is also known. Asymptomatic spreading of this virus is almost non existent and when asymptomatic cases are identified the subjects have all tested as not being infectious. Yet governments worldwide still persist with suppression until vaccination policies based on the assumption that no pre-existing community immunity exists. Ignoring the known pathology of this virus.
OK, but for the third and final time, how is the discussion regarding asymptomatic infectious spreaders relevant outside the realm of academic interest?
In other words, in what way would the measures taken so far be any different if we knew for fact that people are only infectious when they also have other symptoms, e.g. coughing and sneezing, or headaches, or fatigue, or loss of smell or taste, or other typical COVID-19 symptoms?
And, on a side note, you seem to be confounding 'previous immunity' with 'asymptomatic spreading'.
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